According to research results published by the American Journal of Physiology University of Florida College of Medicine Researchers, common sweeteners may be the cause of the recent obesity epidemic in the country.
The study showed that ingesting too much fructose (a simple reducing sugar found in many foods) heightens one’s risk of suffering from leptin resistance when compare with a diet high in fats and calories.
Leptin is a hormone that performs a sort of balancing act in our bodies. It checks the balance between how much we eat and how much energy we burn. So you can imagine that being leptin resistant is not good as this removes our bodies’ natural way of trying to balance incoming and outgoing energy. Without the hormone, we would be more likely to eat and eat and eat.
The research group is quick to point out that you should NOT stay away from eating fruit, which is of course a well know source of fructose. Instead, “it is the consumption of table sugar and high-fructose corn syrup, added to many foods these days, which is causing people to eat much more fructose than ever before”.
The study was conducted on rats, which were divided into two groups. Both groups were administered basically the same diet over six months with only one change, one of the groups were given high doses of fructose. At the end of the research period, the rats on the high-fructose intake diet showed higher levels of triglycerides in their blood steams.
The rats were done injected with leptin and the researches found that those in the ‘normal diet’ group were reducing their food intake. Those on the high-fructose diet, however, became leptin-resistant and did not decrease their food intake.
The study is one of the first to link fructose consumption to leptin resistance. Initially, leptin resistance was thought of as a result of being obese. However, as the study showed, one can be leptin resistant without being overweight first. (In fact, being leptin resistance can lead to obesity.)
“Usually, leptin resistance is associated with obesity, but in this case, leptin resistance developed without obesity. This was very surprising,” according to research team member Alexandra Shapiro.
Source: oneindia
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